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    CIALIS is only for men over the age of 18, including men with diabetes or who have undergone prostatectomy. CIALIS for the Treatment of Symptoms of BPH BPH is a condition that happens in men, where the prostate gland enlarges which can cause urinary symptoms. CIALIS for the Treatment of ED and Symptoms.

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    CIALIS 5 mg For Once Daily Use For Benign Prostatic Hyperplasia (BPH) The efficacy and safety of CIALIS for once daily use for the treatment of the signs and symptoms of BPH was evaluated in 3 randomized, multinational, double-blinded, placebo-controlled, parallel-design, efficacy and safety studies of 12 weeks duration.

    Benefits of Cialis for BPH – Approved by FDA. A lot of scientific works are devoted to the importance of erectile dysfunction (ED) drugs in prolonged BPH treatment. Commonly prescribed to treat ED, Cialis belongs to the class of PDE5 inhibitors and successfully reduces lower urinary tract symptoms.

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    Cialis For Bph

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    INDICATIONS Erectile Dysfunction. CIALIS® is indicated for the treatment of erectile dysfunction (ED). Benign Prostatic Hyperplasia. CIALIS is indicated for the treatment of the signs and symptoms of benign prostatic hyperplasia (BPH).


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    The cause is unclear.
    Treatment options including lifestyle changes, medications, a number of procedures, and surgery.
    About 105 million people are affected globally.
    BPH is the most common cause of lower urinary tract symptoms (LUTS), which are divided into storage, voiding, and symptoms which occur after urination.
    Storage symptoms include the need to urinate frequently, waking at night to urinate , urgency (compelling need to void that cannot be deferred), involuntary urination , including involuntary urination at night, or urge incontinence (urine leak following a strong sudden need to urinate). 6 Voiding symptoms include urinary hesitancy (a delay between trying to urinate and the flow actually beginning), intermittency (not continuous), 7 involuntary interruption of voiding, weak urinary stream, straining to void, a sensation of incomplete emptying, and uncontrollable leaking after the end of urination.
    Bladder outlet obstruction (BOO) can be caused by BPH. 12 Symptoms are abdominal pain, a continuous feeling of a full bladder, frequent urination, acute urinary retention (inability to urinate), pain during urination (dysuria), problems starting urination (urinary hesitancy), slow urine flow, starting and stopping (urinary intermittency), and nocturia.
    BPH can be a progressive disease, especially if left untreated.
    Most experts consider androgens ( testosterone and related hormones ) to play a permissive role in the development of BPH. This means that androgens must be present for BPH to occur, but do not necessarily directly cause the condition.
    This is supported by evidence suggesting that castrated boys do not develop BPH when they age.

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    In an unusual study of 26 eunuchs from the palace of the Qing dynasty still living in Beijing in 1960, the prostate could not be felt in 81% of the studied eunuchs.
    The average time since castration was 54 years (range, 41–65 years). On the other hand, some studies suggest that administering exogenous testosterone is not associated with a significant increase in the risk of BPH symptoms, so the role of testosterone in prostate cancer and BPH is still unclear.
    Further randomized controlled trials with more participants are needed to quantify any risk of giving exogenous testosterone.
    DHT is ten times more potent than testosterone because it dissociates from the androgen receptor more slowly.
    Testosterone promotes prostate cell proliferation, 18 but relatively low levels of serum testosterone are found in patients with BPH. 19 20 One small study has shown that medical castration lowers the serum and prostate hormone levels unevenly, having less effect on testosterone and dihydrotestosterone levels in the prostate.
    While there is some evidence that estrogen may play a role in the cause of BPH, this effect appears to be mediated mainly through local conversion of androgens to estrogen in the prostate tissue rather than a direct effect of estrogen itself.

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